How Sugar intake can increase the risk of Alzheimer’s
From increasing blood sugar levels to impacting the risk of Alzheimer’s disease, sugar does more harm than good. In this episode, we will explore how sugar consumption can increase the risk of Alzheimer’s disease.
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High blood sugar levels increase the risk of Alzheimer’s disease. Although specific cellular mechanisms of link were unknown, researchers have now revealed various brain cells interact with high levels of sugar to increase the risk of Alzheimer’s disease.
Krisna Hanks, a certified health coach, recently twitted the following with a link to an article that speaks about how sugar intake could increase Alzheimer’s risk.
The tweet read:
“Sugar is not your brain’s friend”
Scientists at Wake Forest University School of Medicine have recently found that increased blood sugar levels directly contribute to the accumulation of amyloid plaque in the brain, which increases the risk of Alzheimer’s disease significantly.
Before moving on, let us first understand what is Alzheimer’s disease?
A brain disorder, Alzheimer’s disease begins slowly by destroying memory and thinking skills and eventually wipes out memory making everyday tasks difficult. The disease, in most cases, strikes in old age; however, in rare cases, it can occur between the 30s and mid-60s of a person.
Discovered in 1906 by Dr. Alois Alzheimer, the disease can completely turn an active person into someone who is cut off from the rest of the world.
How is sugar tied to Alzheimer’s plaque formation?
The scientists associated with the research found that ATP- sensitive potassium, a brain channel associated with regulating brain cell activity based on energy levels, led to a greater formation of amyloid plaques in a mouse model when given sugar water than in normal water.
An article published on MedicalNewsToday mentions the following and I quote:
“These findings provide evidence for a potential direct impact of sugar intake and blood glucose levels on the formation and production of amyloid plaques, a crucial characteristic of Alzheimer’s disease.”
“Investigating the underlying mechanisms, the research team identified a group of metabolic sensors called ATP-sensitive potassium channels, or KATP channels, present in neurons. These channels act as a connection between metabolic changes, neuronal activity, and the production of beta-amyloid in the brain,” the article further mentions.
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